Acuity enhancement

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Visual acuity enhancement by StingrayZ - This animation serves as a replication of visual acuity enhancement, which is a common psychedelic effect. It demonstrates the general differences between normal vision and acuity enhancement by shifting between the two states. There is also a subtle amount of visual drifting within this replication.

Acuity enhancement is defined as a heightening of the clearness and clarity of vision. This results in the visual details of the external environment becoming sharpened to the point where the edges of objects become perceived as extremely focused, clear, and defined. The experience of acuity enhancement can be likened to bringing a camera or projector lens that was slightly blurry into focus. At its highest level, a person may experience the ability to observe and comprehend their entire visual field simultaneously, including their peripheral vision. This is in contrast to the default sober state where a person is only able to perceive the small area of central vision in detail.[1]

While under the influence of this effect, it is common for people to suddenly notice patterns and details in the environment they may have never previously noticed or appreciated. For example, the complexity and perceived beauty of the visual input often become apparent when looking at sceneries, nature, and everyday textures.

Acuity enhancement is often accompanied by other coinciding effects such as color enhancement and pattern recognition enhancement.[2][3] It is most commonly induced under the influence of mild dosages of psychedelic compounds, such as LSD, psilocybin, and mescaline. However, it can also occur to a lesser extent under the influence of certain stimulants and dissociatives such as MDMA or 3-MeO-PCP.

Image examples

Analysis

It is thought that a fundamental feature of information-processing dysfunction in both hallucinogen-induced states and schizophrenia-spectrum disorders is the inability of these people to screen out, inhibit, filter, or gate irrelevant stimuli and to attend selectively to more important features of the environment.[4][5][6]

The CSTC model of the brain posits that the thalamus plays a key role in controlling or gating external sensory information to the conscious faculties and is thereby fundamentally involved in the regulation of a person's awareness and attention.[7][8][9][10] The interruption of psychedelics to these neural pathways that inhibit the sensory gating systems[11][10] may, therefore, result in an enhanced availability of sensory information which is usually filtered out by these systems. This process is likely also involved in the various visual, tactile, and auditory enhancements which commonly occur when under the influence of a psychedelic experience.

Psychoactive substances

Compounds within our psychoactive substance index which may cause this effect include:


Experience reports

Anecdotal reports which describe this effect within our experience index include:


See also

External links

References

  1. ↑ Sardegna, Jill; Shelly, Susan; Rutzen, Allan Richard; Scott M Steidl (2002). The Encyclopedia of Blindness and Vision Impairment. Infobase Publishing. p. 253. ISBN 978-0-8160-6623-0. Retrieved 30 November 2014.
  2. ↑ Papoutsis, Ioannis; Nikolaou, Panagiota; Stefanidou, Maria; Spiliopoulou, Chara; Athanaselis, Sotiris (2014). "25B-NBOMe and its precursor 2C-B: modern trends and hidden dangers". Forensic Toxicology. 33 (1): 1–11. doi:10.1007/s11419-014-0242-9. ISSN 1860-8965. 
  3. ↑ Bersani, Francesco Saverio; Corazza, Ornella; Albano, Gabriella; Valeriani, Giuseppe; Santacroce, Rita; Bolzan Mariotti Posocco, Flaminia; Cinosi, Eduardo; Simonato, Pierluigi; Martinotti, Giovanni; Bersani, Giuseppe; Schifano, Fabrizio (2014). "25C-NBOMe: Preliminary Data on Pharmacology, Psychoactive Effects, and Toxicity of a New Potent and Dangerous Hallucinogenic Drug". BioMed Research International. 2014: 1–6. doi:10.1155/2014/734749. ISSN 2314-6133. 
  4. ↑ "Preliminary evidence of an association between sensorimotor gating and distractibility in psychosis". The Journal of Neuropsychiatry and Clinical Neurosciences. 8 (1): 60–66. 1996. doi:10.1176/jnp.8.1.60. ISSN 0895-0172. 
  5. ↑ McGhie, Andrew; Chapman, James (1961). "Disorders of attention and perception in early schizophrenia". British Journal of Medical Psychology. 34 (2): 103–116. doi:10.1111/j.2044-8341.1961.tb00936.x. ISSN 0007-1129. 
  6. ↑ Vollenweider, F. (2007). "Advances and Pathophysiological Models of Hallucinogenic Drug Actions in Humans: A Preamble to Schizophrenia Research". Pharmacopsychiatry. 31 (S 2): 92–103. doi:10.1055/s-2007-979353. ISSN 0176-3679. 
  7. ↑ Goddard, A. W.; Charney, D. S. Toward an integrated neurobiology of panic disorder. J. Clin. Psychiatry 58(suppl. 2):4–11; 1997. https://www.ncbi.nlm.nih.gov/pubmed/9078988
  8. ↑ Steriade, M.; Descheˆnes, M. Cellular thalamic mechanisms. In: Bentivoglio, M.; Spreafico, R., eds. Intrathalamic and brainstem-thalamic networks involved in resting and alert state. Amsterdam: Elsevier; 1988:37–62. https://ci.nii.ac.jp/naid/10017402609/en/
  9. ↑ Steriade, M; McCormick, D.; Sejnowski, T. (1993). "Thalamocortical oscillations in the sleeping and aroused brain". Science. 262 (5134): 679–685. doi:10.1126/science.8235588. ISSN 0036-8075. 
  10. ↑ 10.0 10.1 Vollenweider, Franz X; Geyer, Mark A (2001). "A systems model of altered consciousness: integrating natural and drug-induced psychoses". Brain Research Bulletin. 56 (5): 495–507. doi:10.1016/S0361-9230(01)00646-3. ISSN 0361-9230. 
  11. ↑ Vollenweider F. (1998). Recent advances and concepts in the search for biological correlates of hallucinogen-induced altered states of consciousness. Heffter Rev. Psychedel. Res. 1, 21–32. https://ci.nii.ac.jp/naid/10019112167/
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